headerphoto

Environmental Endocrinology Laboratory (EEL)

John McLachlan, Ph.D.

John McLachlan Dr. McLachlan received his B.A. degree in Liberal Arts from the Johns Hopkins University and a doctoral degree in pharmacology from the George Washington University.

Before coming to Tulane and Xavier in 1995, he spent the previous two decades at the National Institute of Environmental Health Sciences (NIEHS), the world’s largest and most comprehensive center for research on the effects of environmental factors on human health. While at NIEHS, Dr. McLachlan’s scientific contributions led to his discovery of the mechanisms by which environmental chemicals alter fetal development. Dr. McLachlan was named Scientific Director of the NIEHS in 1989.

In his first five years in New Orleans, Dr. McLachlan established a Program in the Environment and Women’s Health, formed the nation’s first Center in Environmental Astrobiology, and initiated the Mississippi River Interdisciplinary Research Program.

Dr. McLachlan’s scientific findings and thoughts have been published in over 150 journal articles, 50 book chapters and 5 edited books. He presents, on average, 30 invited lectures per year and has provided scientific counsel to numerous government agencies, most recently, the European Parliament.

His pioneering research on environmental chemicals that mimic the female hormone, estrogen, has established a new field of research called environmental signaling or endocrine disruption. With his leadership, the CBR has become the internationally recognized focal point for research and communication in this important area of environmental study.

Dr. McLachlan’s wide range of research interests is reflected in his community service and awards. He has worked to facilitate environmental literacy among New Orleans school children. His interests in the environment and fetal development led to his appointment as a board member of the Children’s Environmental Health Network. He was presented a lifetime achievement award by the consumers’ organization, DES Action, on behalf of the approximately 4 million women exposed to diethylstilbestrol (DES) during pregnancy. In May 2000, he received the “Hero of the Year” award from the Breast Cancer Fund.

In recognition for his vision and skills, Dr. McLachlan was awarded the prestigious NIH Directors Award just before coming to New Orleans, and in April 1997, he, along with Tiger Woods and George W. Bush, was selected by Newsweek magazine for the “Century Club: One of the 100 People to Watch as America Moves into the Next Millennium.”

John McLachlan's Environmental Endocrinology Laboratory utilizes cutting edge techniques to study environmental signaling. The major area of concentration for the lab's work is environmental estrogens – natural and synthetic chemicals that interact with the estrogen receptor. Lab members examine the interaction of environmental chemicals with other steroid receptors, as well as cellular signaling pathways. Model systems used in these studies include breast and endometrial cancer cell lines. The function of these compounds in animal systems is also examined.

Current foci include:

  • Phytochemical signaling from legume roots to Rhizobium soil bacteria, which initiates the communication central to symbiosis
  • Investigation of the mechanism by which peptide hormone-generated cell signaling pathways function to regulate estrogen receptor activation and gene expression
  • The role of estrogenic and anti-estrogenic flavonoid photochemicals in the regulation of the estrogen receptor function
  • The effect of environmental chemicals (pesticides, plasticizers, and phytochemicals) on cell-signaling and AP-1 activity
  • The physiological mechanisms by which the brain controls behavior
  • The behavioral effects of phytoestrogens on an aging central nervous system
  • The breakdown process involved in aromatic contaminants and petrochemical remediation

  John McLachlan CV  Curriculum Vitae        John McLachlan PubMed  Publications


Selected Papers

Year

Authors

Title

Journal

2009

Myers, vom Saal, Akingbemi, Arizono, Belcher, Colborn, Chahoud, Crain, Farabollini, Guillette, Hassold, Ho, Hunt, Iguchi, Jobling, Kanno, Laufer, Marcus, McLachlan, Nadal, Oehlmann, Olea, Palanza, Parmigiani, Rubin, Schoenfelder, Sonnenschein, Soto, Talsness, Taylor, N. Vandenberg, Vandenbergh, Vogel, WatsoG. n, Welshons, Zoeller

Why Public Health Agencies Cannot Depend on Good Laboratory Practices as a Criterion for Selecting Data: The Case of Bisphenol A

Environmental Health Perspectives

2009

Boue´, Tilghman, Elliott, Zimmerman, Williams, Payton-Stewart, Miraflor, Howell, Shih, Carter-Wientjes, Segar, Beckman, Wiese, Cleveland, McLachlan, Burow

Identification of the Potent Phytoestrogen Glycinol in Elicited Soybean (Glycine max)

Endocrinology

2009

Rhodes, Muir, Elliott, Guillot, Antoon, Penfornis, Tilghman, Salvo, Fonseca, Lacey, Beckman, McLachlan, Rowan, Pochampally, Burow

Adult human mesenchymal stem cells enhance breast
tumorigenesis and promote hormone independence

Breast Cancer Res Treat

2009

Nierth-Simpson, Martin, Chiang, Melnik, Rhodes, Muir, Burow, and McLachlan

Human Uterine Smooth Muscle and Leiomyoma Cells Differ in Their Rapid 17!-Estradiol Signaling:
Implications for Proliferation

Endocrinology

2009

Bratton, Frigo, Vigh-Conrad, Fan, Wadsworth, McLachlan, and Burow

Organochlorine mediated potentiation of the general coactivator p300 through p38 mitogen-activated protein kinase

Carcinogenesis

2008

Crain, Janssen, Edwards, Heindel, Ho, Hunt, Iguchi, Juul, McLachlan, Schwartz, Skakkebaek, Soto, Swan, Walker, Woodruff, Woodruff, Giudice, and Guillette

Female reproductive disorders: the roles of endocrine-disrupting compounds and developmental timing

Fertility and Sterility

2008

Fox, Burow, McLachlan , Miller

Detecting ligands and dissecting nuclear receptor-signaling pathways using recombinant strains of the yeast Saccharomyces cerevisiae

Nature Protocols

2007

Wetherill, Akingbemi, Kanno, McLachlan, Nadal, Sonnenschein, Watson, Zoeller, Belcher

In vitro molecular mechanisms of bisphenol A action

Reproductive Toxicology

2007

vom Saal, Akingbemi, Belcherc, Birnbaum, Crain, Eriksen, Farabollini, Guillette, Hauser, Heinde, Shuk-Mei Ho, Hunt, Iguchi, Jobling, Kanno, Keri, Knudsen, Laufer, LeBlanc, Marcus, McLachlan, Myers, Nadal, Newbold, Olea, Prins, Richter, Rubin, Sonnenschein, Soto, Talsness, Vandenbergh, Vandenberg, Walser-Kuntz, Watson, Welshons, Wetherill and Zoeller

Chapel Hill bisphenol A expert panel consensus statement: Integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure

Reproductive Toxicology

2007

Fox, Gulledge, Engelhaupt, Burow, and McLachlan

Pesticides reduce symbiotic efficiency of nitrogen-fixing rhizobia and host plants

Proc Natl Acad Sci U S A

2006

Crews and McLachlan

Epigenetics, Evolution, Endocrine Disruption, Health, and Disease

Endocrinology

2006

McLachlan, Nierth-Simson, Martin

Endocrine disrupters and female reproductive health

Best Practice & Research Clinical Endocrinology and Metabolism

2006

Guillette, Conard, Lares, Aguilar, McLachlan, Guillette

Altered breast development in young girls from an agricultural environment

Environmental Health Perspectives

2006

Frigo, Basu, Nierth-Simpson, Weldon, Dugan, Elliott, Collins-Burow, Salvo, Zhu, Melnik, Lopez, Kushner, Curiel, Rowan, McLachlan, Burow

p38 MAPK Stimulates, Estrogen-Mediated transcription and proliferation through the phosphorylation and potentiation of the p160 coactivator GRIP1

Molecular Endocrinology

2006

McLachlan

Commentary: Prenatal exposure to DES: a continuing story

International Journal of Epidemiology

2005

Frigo, Vigh, Struckhoff, Elliott, Beckman, Burow, McLachlan

Xenobiotic-induced TNF-alpha expression and  apoptosis through the p38 MAPK signaling pathway

Toxicology Letters

2004

Frigo, Tang, Beckman, Scandurro, Alam, Burow, McLachlan

Mechanism of AP-1 mediated gene expression by select organochlorines through the p38 MAPK pathway

Carcinogenesis

2004

Fox, Starevic, Jones, Burow, McLachlan

Phytoestrogen Signaling and Symbiotic Gene Activation Are Disrupted by Endocrine-Disrupting Chemicals

Environmental Health Perspectives

2003

Li, Chiang, Richard-Davis, Barrett, McLachlan

DNA hypomethlyation and imbalanced expression of DNA methyltransferases in uterine leiomyomas

Gynecologic Oncology

2003

Li, Hansman, Newbold, Davis, McLachlan, Barrett

Neonatal diethylstilberstrol exposure induces persistent elevation of c-fos expression and hypomethylation in its exon-4 mouse uterus

Molecular Carcinogenesis

2003

Li, Hursting, Davis, McLachlan, Barrett

Environmental exposure, DNA Methlyation, and Gene Regulation

Annals of the NY Academy of Sciences

2003

Boue, Wiese, Nehls, Burow, Elliott, Carter-Wientjes, Shih, McLachlan, Cleveland

Evaluation of the Estrogenic Effects of Legume Extracts Containing Phytoestrogens

Journal of Agricultural and Food Chemistry

2002

Frigo, Duong, Melnik, Schief, Collins-Burow, Pace, McLachlan, Burow

Flavonoid Phytochemicals Regulate Activator Protein-1 Signal Transduction Pathways in Endometrial and Kidney Stable Cell Lines

Journal of Nutrition

2002

Frigo, Burow, Mitchell, Chiang, McLachlan

DDT and Its Metabolites Alter Gene Expression in Human Uterine Cell Lines Through Estrogen Receptor-Independent Mechanisms

Environmental Health Perspectives

2001

O'Neil, Burow, Green, McLachlan, Henson

Effects of estrogen on leptin gene promoter activation in MCF-7 breast cancer and JEG-3 chorocarcinoma cells

Molecular and Cellular Endocrinology

2001

Oberdorster, Clay, Cottam, Wilmot, McLachlan, Milner

Common phytochemicals are ecdysteroid agonists and antagonists: a possible evolutionary link between vertebrate and invertebrate steroid hormones

Journal of Steroid Biochemistry and Molecular Biology

2001

McLachlan, Burow, Chiang, Li

Gene imprinting in developmental toxicology; a possilbe interface between physiology and pathology

Toxicology Letters

2001

McLachlan

Environmental Signaling: what embryos and evolution teach us about endocrine disrupting chemicals

Endocrine Reviews

2001

Li, McLachlan

Estrogen-associated genes in uterine leiomyoma

Annals of the NY Academy of Sciences

2001

Li, Chiang, Richard-Davis, Williams, Wilson, McLachlan

Expression of Wnt7a is inversely associated with the expression of estrogen receptor alpha in human uterine leiomyoma

Journal of Clinical Endocrinology and Metabolism

2001

Li, Ma, Chiang, Burow, Newbold, Negishi, Barrett, McLachlan

Promoter CpG methylation of Hox-a 10 and Hox-a 11 in mouse uterus not altered upon neonatal diethylstilbestrol exposure

Molecular Carcinogenesis

2001

Cheek, Brouwer, Carroll, Manning, McLachlan, Brouwer

Experimental evaluation of vitellogenin as a predictive biomarker for reproductive disruption

Environmental Health Perspectives

2001

Burow, Boue, Collins-Burow, Melnik, Duong, Carter-Wientjes, Li, Wiese, Cleveland, McLachlan

Phytochemical glyceollins, isolated from soy, mediate antihormonal effects through estrogen receptor alpha and beta

Journal of Clinical Endocrinology and Metabolism

2001

Burow, Weldon, Tang, McLachlan, Beckman

Oestrogen-mediated suppression of tumour necrosis alpha-induced apoptosis in MCF-7 cells: subversion of Bcl-2 by anti-oestrogens

Journal of Steroid Biochemistry and Molecular Biology

2000

Newbold, Hanson, Jefferson, Bullock, Haseman, McLachlan

Proliferative lesions and reproductive tract tumors in male descendants of mice exposed developmentally to diethylstilbestrol

Carcinogenesis

1999

Burow, Tang, Collins-Burow, Krajewski, Reed, McLachlan, Beckman

Effects of environmental estrogens on tumor necrosis factor alpha mediated apoptosis in MCF-7 cells

Carcinogenesis

1997

Klotz, Ladlie, Vonier, McLachlan, Arnold

o,p'-DDT and its metabolites inhibit progesterone-dependent responses in yeast and human cells

Molecular and Cellular Endocrinology

1978

Korach, Metzler, McLachlan

Estrogenic activity in vivo and in vitro of some diethlystilbestrol metabolites and analogs

Proceedings of the National Academy of Sciences